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alpha-mannosidosis

MIM.248500 19cen-q12

Alpha-mannosidosis is an autosomal recessive lysosomal storage disease of glycoprotein catabolism caused by a deficiency of lysosomal alpha-mannosidase activity. The disorder shows a wide range of clinical phenotypes, from a severe infantile form (type I), which is fatal at less than 3 to 8 years of age, to a less severe juvenile- or adult-onset form (type II), which ultimately shows progressive neurologic degeneration. - Wide phenotypic variability. Alpha-mannosidosis is caused by mutation in the gene encoding alpha-mannosidase, class 2B1 (MAN2B1) (MIM.609458).

Synopsis

- growth retardation in severe cases
- macrocephaly
- Ffat occiput
- coarse facial features
- broad forehead
- frontal bossing
- midface hypoplasia
- prognathism
- sensorineural deafness
- large ears
- epicanthal folds
- heavy eyebrows
- lenticular ’spoke-like’ opacities
- retinal degeneration in adult patients, progressive
- impaired smooth pursuit in adult patients
- nystagmus in adult patients
- flat nose
- macroglossia
- gingival hypertrophy
- widely-spaced teeth
- pectus carinatum
- thick, undertubulated ribs
- hepatomegaly
- splenomegaly
- inguinal hernia (less common)
- dysostosis multiplex
- thickening of the calvaria
- abnormal vertebral bodies (ovoid, flat, beaked)
- increased vertebral height
- thoracolumbar gibbus deformity
- spondylolithesis
- bowed femurs
- hypertrichosis
- Low anterior hairline
- anterior hair whorl
- heavy eyebrows
- delayed psychomotor development
- mental retardation from rapid mental regression
- hypotonia
- impaired speech
- nerve cells show ballooning with membrane-bound vesicles with reticulogranular pattern
- corticospinal tract disease in lower limbs (in adulthood)
- spinocerebellar tract disease in lower limbs (in adulthood)
- gait ataxia
- limb ataxia
- spasticity
- extensor plantar responses
- dysarthria
- hyperreflexia
- nystagmus
- impaired smooth pursuit ocular movements
- MRI shows cerebellar atrophy (in adulthood)
- white matter changes may occur (in adulthood)
- vacuolated lymphocytes in blood
- recurrent bacterial infections
- recreased serum immunoglobulin

LABORATORY

- Increased urinary mannose-containing oligosaccharides
- Decreased lysosomal alpha-mannosidase activity in plasma and leukocytes

Types

- Type I is infantile-onset, severe, death usually by age 3 to 10 years
- Type II is less-severe, juvenile- or adult-onset

Etiology

Alpha-mannosidosis is caused by mutation in the gene encoding alpha-mannosidase, class 2B1 (MAN2B1) (MIM.609458).