Microscopical anomalies

Articles

• metaplasia
  11 July 2003

  Definition: Metaplasia is the reversible replacement of one differentiated cell type with another differentiated cell type.
  Metaplasia causes precursor cells to change their cell fate, and do not change existing differentiated cells.
  Metaplasia is reversible and usually occurs in response to chronic irritation and inflammation and allows for substitution of cells that are better able to survive under circumstances in which a more fragile cell type might succumb.
  Metaplasia is a (...)

• hypertrophy
  3 June 2003

  Definition: Hypertrophy refers to an increase in the size of cells, resulting in an increase in the size of the organ.
  Pathogenesis
  The hypertrophied organ has no new cells, just larger cells. The increased size of the cells is due not to cellular swelling but to the synthesis of more structural components.
  Cells capable of division may respond to stress by undergoing both hyperplasia and hypertrophy, whereas in nondividing cells (e.g., myocardial fibers), hypertrophy occurs.
  Nuclei (...)

• abscess
  9 June 2004

  abcesses
  Definition: Abscesses are localized collections of purulent inflammatory tissue caused by suppuration buried in a tissue, an organ, or a confined space. They are produced by deep seeding of pyogenic bacteria into a tissue.
  Abscesses have a central region that appears as a mass of necrotic leukocytes and tissue cells. There is usually a zone of preserved neutrophils around this necrotic focus, and outside this region vascular dilation and parenchymal and fibroblastic proliferation (...)

• colonic cryptitis
  29 May 2004

  colonic glandulitis
  Etiology (Examples)
  IBDs Crohn colitis ulcerative colitis
  acute colitis infectious colitis
  See also
  colonic mucosal anomalies colonic cryptic anomalies

• bridging necrosis
  27 January 2004

  Definition: Bridging necrosis is the term given to confluent necrosis linking terminal venules to portal tracts. The term "Bridging necrosis" has been used for necrosis linking any of the vascular structures, but it is now more often restricted to the linking of terminal hepatic venules (centrilobular veins) to portal tracts (central-portal bridging necrosis).
  Bridging necrosis describes the location rather than the type of necrosis. It usually results from extensive necrosis of confluent (...)

• colonic tuberculoid granuloma
  18 October 2004

  Etiology
  colonic Crohn disease
  tuberculous colitis
  colonic sarcoidosis

• multifocal cystic changes in Langerhans islets
  26 March 2007

  Etiology (Examples)
  Johanson-blizzard syndrome (#17378628#)
  References
  Hoffman WH, Lee JR, Kovacs K, Chen H, Yaghmai F. Johanson-blizzard syndrome: autopsy findings with special emphasis on hypopituitarism and review of the literature. Pediatr Dev Pathol. 2007 Jan-Feb;10(1):55-60. PMID: #17378628#

• pancreatic fibrosis
  29 March 2005

  Etiology (Exemples)
  pancreatitis post-acute pancreatitis chronic pancreatitis
  pancreatic secretion anomalies Cystic fibrosis pancreatic cystic fibrosis
  anomalies of pancreatic organogenesis (pancreatic dysplasia) Jeune syndrome (#3227005#)
  See also
  fetal pancreatic fibrosis

• colonic cryptic fibrosis
  5 December 2004
• Paneth cell metaplasia
  27 June 2003

  See also : Paneth cell / Paneth cells
  Images
  Paneth cell metaplasia https://twitter.com/dmlarsonpath1/status/861241980147421188 https://twitter.com/BenMazer/status/804722056512368645
  Etiology
  chronic colitis active colitis inflammatory bowel diseases (IBDs ) Crohn colitis ulcerative colitis diversion colitis (#10632750#)
  Images
  Paneth cell metaplasia https://twitter.com/RikiSotillo/status/843812126804336640
  See also
  metaplasia
  References
  Sandow MJ, Whitehead R. The (...)

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