Mophological syndrome

Articles

• interface hepatitis
  25 May 2004

  piecemeal necrosis

• acute inflammation
  1 June 2004

  Digital cases
  Case 25 (HPC:25) : Acute appendicitis
  Case 94 (HPC:94) : Acute appendicitis
  Case 13 (HPC:13) : Acute synovitis
  Definition: Acute inflammation is a rapid response to an injurious agent that serves to deliver mediators of host defense-leukocytes and plasma proteins-to the site of injury.
  Types - Lesional patterns
  acute inflammation
  congestive inflammation
  cellular acute inflammation
  suppurative inflammation
  gangrenous inflammation
  It can be assessed by the (...)

• appendicitis
  29 December 2003

  Definition: inflammation of appendix.
  Types
  acute appendicitis
  chronic appendicitis granulomatous appendicitis xanthogranulomatous appendicitis
  mural fibrosis of appendix
  Etiology
  infectious appendicitis viral appendicitis adenoviral appendicitis CMV appendicitis bacterial appendicitis Yersinia appendicitis Yersinia enterocolitica appendicitis (#17189675#) appendiceal typhoid appendiceal actinomycosis mycobacterial appendicitis tuberculous appendicitis fungal appendicitis (...)

• chronic active gastritis
  18 October 2005

  CAG, active chronic gastritis

• acute hepatitis
  30 May 2003

  Microscopy
  The essential components of the acute phase of hepatitis are:
  inflammatory-cell infiltration
  hepatocellular damage
  cholestasis
  Kupffer-cell activation
  endotheliitis
  bile-duct damage
  ductular reaction
  hepatocellular regeneration
  hepatocellular damage
  Hepatocellular damage range from minor degrees of cell swelling to cell death. They are accompanied by the inflammatory infiltration described below, reflecting the important role of cellular immunity in the (...)

• massive hepatic necrosis
  8 December 2004

  Massive and submassive hepatic necrosis are the histopathological counterpart of fulminant hepatic failure.
  Etiology
  infections acute viral hepatitis (60 to 75%)
  intoxications (drugs and toxins) (20%) drug-induced hepatic necrosis toxin-induced hepatic necrosis
  severe hepatic ischemia
  hepatic metabolic diseases Wilson disease hereditary tyrosinemia galactosemia hereditary fructose intolerance
  congenital hemochromatosis
  acute auto-immune (...)

• canalicular cholestasis
  8 December 2004

  Canalicular cholestasis takes the form of bile plugs (bile thrombi) in dilated canaliculi.
  There is often brown or yellow pigment in nearby hepatocytes and Kupffer cells, but the distinction of this pigment from others such as lipofuscin and ceroid is not a serious practical problem; this is because the presence of bile in the canaliculi makes the diagnosis of cholestasis obvious.
  In general, cholestasis should only be diagnosed with great caution in the absence of bile plugs in (...)

• intestinal villous atrophy
  15 December 2003

  atrophic villi, flat villi
  Synopsis
  atrophic and flat intestinal villi
  Types (according to extension)
  focal villous atrophy
  diffuse villous atrophy
  Types (according to crypt appearance)
  hyperplastic villous atrophy
  hypoplastic villous atrophy (with crypt hyperplasia)
  Types (according to inflammation)
  villous atrophy with mucosal inflammation
  villous atrophy without mucosal inflammation
  Etiology
  hyperplastic villous atrophy (with crypt hyperplasia) coeliac disease (...)

• gastric mucosal atrophy
  7 March 2005

  Definition: Atrophy means wasting, with resulting reduction in size or amount of an organ, tissue, or cell type.
  Atrophy of the stomach affects primarily the glands, which become sparse and small. In atrophy of the gastric corpus and fundus, parietal and chief cells disappear from the oxyntic glands, and the residual glands may undergo pseudopyloric metaplasia.
  Mucosal atrophy can be noted grossly, especially in advanced, diffuse atrophy of oxyntic mucosa in the fundus and corpus where (...)

• lymphoepithelial sialadenitis
  5 November 2007

  LESA, myoepithelial sialadenitis, MESA, salivary benign lymphoepithelial lesion,
  Definition: LESA is found in virtually all patients with Sjögren’s syndrome but may occur without Sjogren’s syndrome. It is associated with lymphoid infiltration of salivary gland tissue, producing atrophy and destruction of the acini.
  Myoepithelial sialadenitis of the salivary gland is associated with an increased risk of lymphoma, but criteria that predict its development have not been clearly defined. (...)

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